yap1 inhibitor verteporfin (MedChemExpress)
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yap1 inhibitor verteporfin
Yap1 Inhibitor Verteporfin, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 98/100, based on 346 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/yap1+inhibitor+verteporfin/Verteporfin/pm42048035-93-7-14
Average 98 stars, based on 346 article reviews
Yap1 Inhibitor Verteporfin, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 98/100, based on 346 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/yap1+inhibitor+verteporfin/Verteporfin/pm42048035-93-7-14
Average 98 stars, based on 346 article reviews
yap1 inhibitor verteporfin - by Bioz Stars,
2026-09
98/100 stars
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Injection:Article Title: CXCL12/CXCR7/β-arrestin1 biased signal promotes epithelial-to-mesenchymal transition of colorectal cancer by repressing miRNAs through YAP1 nuclear translocation. Article Snippet: .. The mice injected with HCT116LV−CXCR7 cells were administered daily with Article Title: CXCL12/CXCR7/β-arrestin1 biased signal promotes epithelial-to-mesenchymal transition of colorectal cancer by repressing miRNAs through YAP1 nuclear translocation. Article Snippet: To establish azoxymethane (AOM)/ dextran sodium sulfate (DSS)-induced inflammatory colonic adenocarcinoma, wild type C57BL/6 mice and Villin-CXCR7 mice (aged at 8 weeks, n = 5) were treated by a single intraperitoneal injection of AOM (10 mg/kg, Sigma, USA) and subsequent oral administration of 1% DSS (MP Biomedicals, USA) in drinking water ad libitum for 7 consecutive days and then returned to normal drinking for 14 days. .. VillinCXCR7 mice were injected intraperitoneally daily with Article Title: CXCL12/CXCR7/β-arrestin1 biased signal promotes epithelial-to-mesenchymal transition of colorectal cancer by repressing miRNAs through YAP1 nuclear translocation Article Snippet: To establish azoxymethane (AOM)/ dextran sodium sulfate (DSS)-induced inflammatory colonic adenocarcinoma, wild type C57BL/6 mice and Villin-CXCR7 mice (aged at 8 weeks, n = 5) were treated by a single intraperitoneal injection of AOM (10 mg/kg, Sigma, USA) and subsequent oral administration of 1% DSS (MP Biomedicals, USA) in drinking water ad libitum for 7 consecutive days and then returned to normal drinking for 14 days. .. Villin-CXCR7 mice were injected intraperitoneally daily with Article Title: CXCL12/CXCR7/β-arrestin1 biased signal promotes epithelial-to-mesenchymal transition of colorectal cancer by repressing miRNAs through YAP1 nuclear translocation Article Snippet: .. The mice injected with HCT116 LV−CXCR7 cells were administered daily with Solvent:Article Title: CXCL12/CXCR7/β-arrestin1 biased signal promotes epithelial-to-mesenchymal transition of colorectal cancer by repressing miRNAs through YAP1 nuclear translocation. Article Snippet: .. The mice injected with HCT116LV−CXCR7 cells were administered daily with Article Title: CXCL12/CXCR7/β-arrestin1 biased signal promotes epithelial-to-mesenchymal transition of colorectal cancer by repressing miRNAs through YAP1 nuclear translocation Article Snippet: .. The mice injected with HCT116 LV−CXCR7 cells were administered daily with Control:Article Title: CXCL12/CXCR7/β-arrestin1 biased signal promotes epithelial-to-mesenchymal transition of colorectal cancer by repressing miRNAs through YAP1 nuclear translocation. Article Snippet: .. The mice injected with HCT116LV−CXCR7 cells were administered daily with Article Title: CXCL12/CXCR7/β-arrestin1 biased signal promotes epithelial-to-mesenchymal transition of colorectal cancer by repressing miRNAs through YAP1 nuclear translocation. Article Snippet: To establish azoxymethane (AOM)/ dextran sodium sulfate (DSS)-induced inflammatory colonic adenocarcinoma, wild type C57BL/6 mice and Villin-CXCR7 mice (aged at 8 weeks, n = 5) were treated by a single intraperitoneal injection of AOM (10 mg/kg, Sigma, USA) and subsequent oral administration of 1% DSS (MP Biomedicals, USA) in drinking water ad libitum for 7 consecutive days and then returned to normal drinking for 14 days. .. VillinCXCR7 mice were injected intraperitoneally daily with Article Title: CXCL12/CXCR7/β-arrestin1 biased signal promotes epithelial-to-mesenchymal transition of colorectal cancer by repressing miRNAs through YAP1 nuclear translocation Article Snippet: To establish azoxymethane (AOM)/ dextran sodium sulfate (DSS)-induced inflammatory colonic adenocarcinoma, wild type C57BL/6 mice and Villin-CXCR7 mice (aged at 8 weeks, n = 5) were treated by a single intraperitoneal injection of AOM (10 mg/kg, Sigma, USA) and subsequent oral administration of 1% DSS (MP Biomedicals, USA) in drinking water ad libitum for 7 consecutive days and then returned to normal drinking for 14 days. .. Villin-CXCR7 mice were injected intraperitoneally daily with Article Title: CXCL12/CXCR7/β-arrestin1 biased signal promotes epithelial-to-mesenchymal transition of colorectal cancer by repressing miRNAs through YAP1 nuclear translocation Article Snippet: .. The mice injected with HCT116 LV−CXCR7 cells were administered daily with Saline:Article Title: CXCL12/CXCR7/β-arrestin1 biased signal promotes epithelial-to-mesenchymal transition of colorectal cancer by repressing miRNAs through YAP1 nuclear translocation. Article Snippet: To establish azoxymethane (AOM)/ dextran sodium sulfate (DSS)-induced inflammatory colonic adenocarcinoma, wild type C57BL/6 mice and Villin-CXCR7 mice (aged at 8 weeks, n = 5) were treated by a single intraperitoneal injection of AOM (10 mg/kg, Sigma, USA) and subsequent oral administration of 1% DSS (MP Biomedicals, USA) in drinking water ad libitum for 7 consecutive days and then returned to normal drinking for 14 days. .. VillinCXCR7 mice were injected intraperitoneally daily with Article Title: CXCL12/CXCR7/β-arrestin1 biased signal promotes epithelial-to-mesenchymal transition of colorectal cancer by repressing miRNAs through YAP1 nuclear translocation Article Snippet: To establish azoxymethane (AOM)/ dextran sodium sulfate (DSS)-induced inflammatory colonic adenocarcinoma, wild type C57BL/6 mice and Villin-CXCR7 mice (aged at 8 weeks, n = 5) were treated by a single intraperitoneal injection of AOM (10 mg/kg, Sigma, USA) and subsequent oral administration of 1% DSS (MP Biomedicals, USA) in drinking water ad libitum for 7 consecutive days and then returned to normal drinking for 14 days. .. Villin-CXCR7 mice were injected intraperitoneally daily with Transfection:Article Title: HIRA promotes osteogenic differentiation of BMSCs and ameliorates osteoporosis by mediating M2 polarization of macrophages through the YAP1/β-catenin pathway. Article Snippet: Extended author information available on the last page of the article Abstract Impaired macrophage polarization is a key factor exacerbating osteoporosis (OP), influencing bone metabolism via modulating the osteogenic differentiation of bone marrow mesenchymal stem cells (BMSCs).. Histone cell cycle regulator (HIRA), an epigenetic regulator, is downregulated in OP.. However, its role in modulating macrophage polarization and BMSCs osteogenic differentiation remains unclear. Concentration Assay:Article Title: LH promotes testosterone synthesis in rooster leydig cells through YAP1/ACSL4/SOAT1 pathway. Article Snippet: Testosterone synthesis in Leydig cells requires precise coordination between LH signaling and cholesterol metabolism, but the mechanisms underlying this regulation remain incompletely understood.. In this study, we reveal a previously unrecognized YAP1-ACSL4 axis essential for LH-induced testosterone production by integrating transcriptomic profiling and functional studies in Leydig cells.. Transcriptomic sequencing identified ACSL4 and YAP1 as key regulators of testosterone synthesis induced by LH. |
